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Psychology News RobotDATE: August 22, 2025 at 04:00PM SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. ** -------------------------------------------------TITLE: Americans broadly agree on what’s “woke,” but partisan cues still shape perceptionsURL: <a href="https://www.psypost.org/americans-broadly-agree-on-whats-woke-but-partisan-cues-still-shape-perceptions/" rel="nofollow noopener" translate="no" target="_blank">https://www.psypost.org/americans-broadly-agree-on-whats-woke-but-partisan-cues-still-shape-perceptions/</a>A new study published in Research and Politics provides insight into how Americans conceptualize the term “woke.” While Democrats, Republicans, and Independents tend to agree on many of the attributes associated with being woke, the study found that partisanship shapes which issues, identities, and policies people associate with the label.Although “woke” has become a widely used political term—especially in conservative media and Republican rhetoric—there remains little clarity about what the average person actually thinks it means. Originally used by Black civil rights activists to encourage vigilance against racial injustice, the term later gained traction in the 2010s during protests against police brutality. But as its popularity grew, so did its ambiguity. Over time, “woke” shifted from signifying a commitment to social justice to being used by the political right as a vague pejorative encompassing a range of progressive causes and behaviors.The researchers aimed to explore whether this rhetorical evolution has altered how people across the political spectrum perceive the term. Specifically, they asked: What does the American public consider to be “woke”? And do definitions differ between Democrats, Republicans, and Independents?To investigate public perceptions of wokeness, the researchers conducted a conjoint survey experiment. They recruited 1,126 participants living in the United States through Lucid/Cint, a platform that provides samples designed to reflect U.S. demographics in terms of age, gender, and race. While not a probability sample, this approach is commonly used in political science experiments to identify opinion patterns.Each participant was shown two lists of items across five trials. The items included political parties, racial and gender identities, professions, products, policies, and public figures—many of which have been described as “woke” in recent media coverage. The task was simple: choose which of the two lists was more “woke,” based on the participant’s own understanding of the term. Across all trials, this yielded over 11,000 individual responses.The researchers deliberately included both items clearly associated with progressive causes—such as Black Lives Matter or pro-choice policies—and items with weaker or more ambiguous ties to wokeness, such as professions, consumer products, and higher education institutions.In addition to the main experiment, participants were also asked questions measuring authoritarian attitudes, such as support for rule-breaking leaders or political violence. These items were used to explore how broader ideological orientations might influence perceptions of wokeness.The study found that certain items were broadly perceived as “woke” across the political spectrum. These included Democratic Party identifiers, transgender people, lesbian identities, Black Americans, the Civil Rights Movement, Black Lives Matter, and pro-choice policies. Items that were consistently viewed as anti-woke included the Republican Party, straight men, Donald Trump, book bans, the Proud Boys, and the Ku Klux Klan.Interestingly, not every political figure fell clearly on either side of the spectrum. For example, Joe Biden, Chuck Schumer, Ron DeSantis, and Matt Gaetz were all perceived as relatively neutral, despite their strong affiliations with their respective parties. Similarly, high-profile right-wing groups such as Moms for Liberty and Antifa were not reliably seen as either woke or anti-woke by respondents.When the researchers examined responses by political affiliation, they found that partisanship played a defining role in how participants judged what was woke. For Republican respondents, items associated with the Democratic Party—regardless of their racial or gender content—were more likely to be seen as woke. Democratic respondents, on the other hand, tended to associate wokeness more with items that represented progressivism on race and gender issues.For instance, Democrats were more likely to say that profiles containing Black Lives Matter, Planned Parenthood, or feminist identifiers were woke. Republicans, by contrast, focused more on party cues. They were more likely to say something was woke if it included prominent Democrats or policies aligned with liberal values.Independents offered a mixed picture. They were somewhat more likely to agree with Democrats when it came to gender progressivism but showed less consistency overall. Their views seemed to borrow selectively from both partisan frameworks.Another layer of analysis revealed subtle but telling gender dynamics. Republican respondents often associated women and female political figures—especially Nancy Pelosi and Alexandria Ocasio-Cortez—with wokeness, while not extending the same judgment to male Democratic figures like Joe Biden. Gender-oriented causes and groups were often more strongly linked to wokeness among Republican and Independent respondents than racial topics were. This suggests that opposition to gender progressivism may be a more potent trigger for anti-woke sentiment on the political right.The results tend to support the idea that the term “woke” has experienced significant conceptual stretching. Once focused on racial injustice, the term now appears to operate as a general label for progressive ideas and identities, particularly those associated with the Democratic Party.But the researchers caution that their forced-choice design may not perfectly reflect how people think about wokeness in real-world settings. Participants had to choose one list as more woke, even if both seemed equally woke—or not woke at all—to them. This may have pushed some respondents toward making artificial distinctions. Future studies could explore more open-ended or nuanced approaches to understanding how people define the term.The study, “What’s woke? Ordinary Americans’ understandings of wokeness,” was authored by Benjamin M. VanDreew, Joseph B. Phillips, B. Kal Munis, and Spencer Goidel.URL: <a href="https://www.psypost.org/americans-broadly-agree-on-whats-woke-but-partisan-cues-still-shape-perceptions/" rel="nofollow noopener" translate="no" target="_blank">https://www.psypost.org/americans-broadly-agree-on-whats-woke-but-partisan-cues-still-shape-perceptions/</a>-------------------------------------------------Private, vetted email list for mental health professionals: <a href="https://www.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">https://www.clinicians-exchange.org</a>Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot <a href="https://mastodon.clinicians-exchange.org/@PTUnofficialBot" class="u-url mention" rel="nofollow noopener" target="_blank">@PTUnofficialBot</a>NYU Information for Practice puts out 400-500 good quality health-related research posts per week but its too much for many people, so that bot is limited to just subscribers. 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Psychology News RobotDATE: August 22, 2025 at 02:00PM SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. ** -------------------------------------------------TITLE: Scientists rewired people’s romantic “type” using a made-up trait—here’s what happened nextURL: <a href="https://www.psypost.org/scientists-rewired-peoples-romantic-type-using-a-made-up-trait-heres-what-happened-next/" rel="nofollow noopener" translate="no" target="_blank">https://www.psypost.org/scientists-rewired-peoples-romantic-type-using-a-made-up-trait-heres-what-happened-next/</a>People often say they have a “type” when it comes to romantic partners. But how do these ideal traits actually influence how we see others—and even who we choose to date? A new study published in Personality and Social Psychology Bulletin offers experimental evidence that ideal partner preferences don’t just reflect who we are drawn to; they also seem to change how we perceive others and where we direct our romantic efforts. The findings suggest that ideals can lead people to see their current partners in a more flattering light and gravitate toward environments that feature partners who match their preferences.There is a large body of research exploring what traits people say they want in a romantic partner—things like kindness, intelligence, or physical attractiveness. Past studies have found that these ideal preferences are linked to important outcomes, such as satisfaction and commitment. But most of this evidence has been correlational, meaning it can’t determine whether ideals cause these outcomes or merely reflect them.To address this gap, researchers Ariana da Silva Frost and Paul Eastwick set out to design an experiment that could directly manipulate people’s ideal partner preferences and observe the resulting effects. Their goal was to determine which of four competing theories best explains how ideals influence perceptions, preferences, and behavior in romantic relationships.Across two large experiments, the researchers tested how people’s ideal partner preferences could be shaped through experience, and how those altered ideals affected their perception of others. Both studies focused on individuals who were attracted to men and were currently in relationships.The team used a gamified approach called “DateFest,” adapted from previous psychological paradigms, to manipulate participants’ preferences for a novel trait. In the first study, they invented a term—“Reditry”—to represent a trait that was actually babyfacedness or youthfulness. In the second study, they dropped the fictional label and used the real word “youthfulness.”During the DateFest game, participants encountered images of 24 potential dating partners and had to decide whether or not to go on a hypothetical date with each one. Unknown to the participants, the game was rigged so that in one version of the study, people with higher Reditry (or youthfulness) tended to lead to more positive experiences, while in another version, this trait was only weakly linked to positive outcomes. This allowed the researchers to manipulate how much participants “learned” to value the trait, based on the outcomes of their choices.After the game, participants answered a variety of questions about their preferences, their current romantic partner, and their perceptions of other people they knew.The first study included 1,639 participants, the majority of whom were women, with an average age of 28. All were currently in romantic relationships and primarily attracted to men. Participants were randomly assigned to experience either a strong or weak version of the Reditry preference manipulation.In the second study, 2,027 participants completed the same tasks, except the manipulated trait was explicitly described as youthfulness rather than using a fabricated label. This study also included additional measures to assess whether the effects could be attributed to participants trying to please the researchers or responding to demand characteristics.In both studies, the researchers assessed four potential outcomes: whether participants perceived their current partner as higher in the target trait, whether the trait influenced their relationship satisfaction, whether it changed how they saw other people in their lives, and whether it influenced hypothetical choices to enter environments with more potential partners who matched the trait.Across both studies, the strongest and most consistent finding was that participants who were led to value Reditry or youthfulness more also came to believe that their current partner possessed more of that trait. This finding supports what the researchers call the “motivated projection” theory—the idea that people see what they want to see in their romantic partners.There was also support for what the researchers term “situation selection.” Participants who came to value the trait more strongly expressed greater interest in joining a hypothetical dating website filled with potential partners high in that trait. This suggests that ideals may influence how people shape their romantic opportunities, not just how they evaluate individuals.The results for the third theory, “perceiver effects,” were more mixed. Participants who were led to value the trait more also tended to believe that their friends and themselves had more of the trait, but this effect did not always extend to strangers or disliked individuals.The weakest support was found for the idea that people are more satisfied in their relationships when their partner matches their ideals—known as the “preference-matching” or trait-weighting model. This effect emerged in the first study when the trait was labeled “Reditry,” but disappeared in the second study when it was labeled “youthfulness.” The researchers suggest this inconsistency may reflect the difficulty of detecting such effects when the trait has clear real-world meaning or is more resistant to reinterpretation.Interestingly, the studies also showed that manipulating how much participants valued a trait could alter their interpretation of that trait. For example, participants who were induced to value youthfulness were more likely to interpret it in a positive way (as energetic or active) rather than a negative one (as immature or childish).But there are limitations to keep in mind. First, both studies focused exclusively on people attracted to men and used male facial stimuli, meaning the results may not generalize to those attracted to women or nonbinary individuals. Second, all participants were in relationships, so the hypothetical nature of some tasks (like the dating website scenario) may have limited their ecological validity.The traits selected for manipulation—babyfacedness and youthfulness—also have unique characteristics that may not apply to all partner ideals. Future research could apply similar methods to other traits such as ambition, kindness, or physical attractiveness.Additionally, while these experiments provide strong evidence that ideals influence perception and decision-making, the underlying psychological mechanisms remain unclear. More work is needed to understand exactly how motivated reasoning and belief formation interact in shaping romantic perceptions.The study, “Experimental Tests of the Role of Ideal Partner Preferences in Relationships,” was authored by Aline da Silva Frost and Paul W. Eastwick.URL: <a href="https://www.psypost.org/scientists-rewired-peoples-romantic-type-using-a-made-up-trait-heres-what-happened-next/" rel="nofollow noopener" translate="no" target="_blank">https://www.psypost.org/scientists-rewired-peoples-romantic-type-using-a-made-up-trait-heres-what-happened-next/</a>-------------------------------------------------Private, vetted email list for mental health professionals: <a href="https://www.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">https://www.clinicians-exchange.org</a>Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot <a href="https://mastodon.clinicians-exchange.org/@PTUnofficialBot" class="u-url mention" rel="nofollow noopener" target="_blank">@PTUnofficialBot</a>NYU Information for Practice puts out 400-500 good quality health-related research posts per week but its too much for many people, so that bot is limited to just subscribers. 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Psychology News RobotDATE: August 22, 2025 at 12:00PM SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. ** -------------------------------------------------TITLE: Dopamine-boosting drug enhances self-control and reduces drinking in people with alcohol use disorderURL: <a href="https://www.psypost.org/dopamine-boosting-drug-enhances-self-control-and-reduces-drinking-in-people-with-alcohol-use-disorder/" rel="nofollow noopener" translate="no" target="_blank">https://www.psypost.org/dopamine-boosting-drug-enhances-self-control-and-reduces-drinking-in-people-with-alcohol-use-disorder/</a>A new clinical trial has found evidence that the dopamine-enhancing drug tolcapone can help improve self-control and reduce alcohol consumption in people with alcohol use disorder. The study, published in Biological Psychiatry: Cognitive Neuroscience and Neuroimaging, found that tolcapone increased activity in brain regions involved in inhibitory control, particularly the inferior frontal gyrus and dorsolateral prefrontal cortex. These changes were associated with improved performance on a self-control task and fewer drinks consumed during the trial.Alcohol use disorder involves both strong motivation to consume alcohol and a reduced ability to inhibit that motivation. Brain circuits that support self-control are thought to be impaired in people with this condition. Specifically, lower dopamine signaling in the prefrontal cortex has been observed in individuals with alcohol use disorder, and this may contribute to the loss of behavioral control.Previous attempts to treat alcohol use disorder with dopamine-related medications have had mixed results, possibly because these drugs influence dopamine levels across broad brain regions. In contrast, tolcapone offers a more targeted approach. It inhibits an enzyme called catechol-O-methyltransferase, which breaks down dopamine primarily in the prefrontal cortex. By increasing dopamine availability specifically in this region, tolcapone may help restore some of the self-regulatory functions that are disrupted in alcohol use disorder.“Alcohol use disorder is a prevalent and devastating disease for which only three medications are currently approved by the U.S. Food and Drug Administration (FDA),” said study author Joseph P. Schacht, an associate professor of psychiatry at the University of Colorado School of Medicine.“These existing medications are believed to work by reducing craving for alcohol and/or changing its acute effects (increasing negative effects or reducing euphoric effects). A core symptom of alcohol use disorder is loss of control over drinking, and we wanted to explore whether a different kind of medication, which might increase behavioral control, might be effective for people with alcohol use disorder. “We conducted a randomized, placebo-controlled trial of tolcapone, an FDA-approved medication for Parkinson’s disease. Tolcapone inhibits the enzyme catechol-O-methyltransferase (COMT), which degrades catecholamine neurotransmitters in the brain. This means that it can transiently increase concentrations of the neurotransmitter dopamine. We often think of dopamine as being related to reward and euphoria, but in the prefrontal cortex, dopamine is critical for many cognitive processes that involve behavioral control. We thought that, by increasing prefrontal dopamine, tolcapone might increase behavioral control among people with alcohol use disorder.”The trial was conducted at the Medical University of South Carolina. Participants were adults aged 21 to 40 who met diagnostic criteria for moderate or severe alcohol use disorder. They were required to drink at least 20 standard drinks per week and were not seeking treatment for their alcohol use. A total of 64 individuals with usable brain imaging data were included in the final analysis.Participants were randomly assigned to receive either tolcapone or a placebo for eight days. Tolcapone dosage was gradually increased to a maximum of 200 mg three times per day. Brain activity was measured using functional magnetic resonance imaging (fMRI) before and after the medication period. The primary task used to measure self-control was the stop-signal task, which requires individuals to inhibit a dominant motor response when signaled.The researchers also tracked participants’ alcohol consumption using a daily self-report tool. To ensure the accuracy of the brain imaging results, several quality control measures were implemented, including controlling for motion during scanning and using different MRI scanner settings as covariates in the analysis.The researchers found that participants who received tolcapone showed improvements in inhibitory control. This was measured by shorter stop-signal reaction times, indicating that they were better able to withhold a response when prompted. They also showed more behavioral adjustment after making errors, another indicator of improved self-regulation. These improvements were not seen in the placebo group.In addition to changes in behavior, tolcapone also increased activity in brain areas involved in self-control, particularly the inferior frontal gyrus and dorsolateral prefrontal cortex. These increases were only observed in the tolcapone group. Notably, individuals who showed greater increases in activation in these areas also tended to report less alcohol consumption during the trial.“We suspected that greater tolcapone-induced prefrontal cortex activation might be related to better performance on the task, but were pleasantly surprised to find that it was also related to less alcohol consumption,” Schacht told PsyPost. “This suggests that tolcapone’s effect on behavioral control in the brain scanner translated to changes in real-world behavior.”The researchers also found changes in how brain regions communicated with each other. Tolcapone increased connectivity between the inferior frontal gyrus and two other brain regions—the anterior insula and anterior cingulate cortex—that are also involved in emotion regulation and cognitive control. These enhanced connections were linked to better task performance and fewer drinks consumed, but again, only in participants who received tolcapone.The results provide evidence that boosting dopamine levels specifically in the prefrontal cortex may help restore self-control in people with alcohol use disorder. This has been a longstanding goal in addiction research, but previous approaches have struggled to achieve this without unintended effects in other parts of the brain.By showing that tolcapone can influence both brain activity and real-world drinking behavior, the findings suggest that targeted dopamine modulation could be a promising therapeutic approach. The inferior frontal gyrus, in particular, emerged as a central hub in the network supporting self-regulation, with its activity and connectivity predicting both task performance and drinking outcomes.“We used brain imaging (functional magnetic resonance imaging) to measure activity in the prefrontal cortex while people engaged in a simple behavioral control task,” Schacht summarized. “We found that tolcapone, relative to placebo, increased activation of a brain region called the dorsolateral prefrontal cortex, and that increased activation of this region was associated with better behavioral control (better performance on the task) and less alcohol consumption during the time people were taking the medication. This suggests that medications like tolcapone that affect prefrontal dopamine might be a novel strategy for treating alcohol use disorder.”While the mechanism appears to be related to dopamine, the researchers caution that other neurotransmitter systems—such as norepinephrine and glutamate—could also be involved. Additional studies will be needed to disentangle these contributions and to examine whether the effects of tolcapone extend beyond self-control to other aspects of alcohol use disorder, such as craving or mood regulation.Although the findings are promising, some limitations should be noted. The sample size, while sufficient for the planned analyses, was relatively small. Larger trials will be needed to confirm these results and assess how long the benefits of tolcapone might last. The study also excluded people with psychiatric comorbidities or other substance use disorders, so the generalizability of the findings to more diverse clinical populations remains uncertain.In addition, the researchers were not able to test genetic differences in how participants responded to the drug. The study was designed to examine whether genetic variation in a dopamine-related gene (COMT rs4680) affected the drug’s impact, but the number of participants in each genetic subgroup was too small to draw firm conclusions. Prior studies suggest that people with certain genetic variants may respond more strongly to tolcapone, and this will be an important area for future research.“Although it is approved for Parkinson’s disease, tolcapone has the potential for liver toxicity, which can be dangerous among people who may have alcohol-associated liver disease,” Schacht noted. “In this study, we used tolcapone under very controlled conditions (people had to have normal liver function to participate, and they took the medication for only 8 days). There are several approved COMT inhibitors that are not toxic to the liver, but they do not cross the blood-brain barrier like tolcapone does. We hope that COMT inhibitors that are brain-penetrant but not hepatotoxic will be developed in the near future.”There is also a need to better understand whether the effects of tolcapone are specific to improving inhibition of alcohol-related impulses or whether they reflect a broader enhancement of executive function. Future studies could compare how the drug affects brain responses to alcohol cues versus non-alcohol-related tasks, or examine whether it changes patterns of decision-making or emotional regulation in everyday life.“We are currently completing a large study in which we are testing tolcapone among people with alcohol use disorder and attention-deficit/hyperactivity disorder,” Schacht said. “This is a population for which improvements in behavioral control might be especially impactful. We hope to publish our findings within the next year.”“More efficacious medications for alcohol use disorder are badly needed. This study was one small step towards a larger goal within the alcohol research community of identifying novel medications to help improve the treatment of this disease.”The study, “Effects of COMT suppression in a randomized trial on the neural correlates of inhibitory processing among people with Alcohol Use Disorder,” was authored by Drew E. Winters and Joseph P. Schacht.URL: <a href="https://www.psypost.org/dopamine-boosting-drug-enhances-self-control-and-reduces-drinking-in-people-with-alcohol-use-disorder/" rel="nofollow noopener" translate="no" target="_blank">https://www.psypost.org/dopamine-boosting-drug-enhances-self-control-and-reduces-drinking-in-people-with-alcohol-use-disorder/</a>-------------------------------------------------Private, vetted email list for mental health professionals: <a href="https://www.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">https://www.clinicians-exchange.org</a>Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot <a href="https://mastodon.clinicians-exchange.org/@PTUnofficialBot" class="u-url mention" rel="nofollow noopener" target="_blank">@PTUnofficialBot</a>NYU Information for Practice puts out 400-500 good quality health-related research posts per week but its too much for many people, so that bot is limited to just subscribers. 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Psychology News RobotDATE: August 22, 2025 at 10:00AM SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. ** -------------------------------------------------TITLE: Prenatal cannabis exposure linked to blunted brain response and psychotic-like symptoms in youthURL: <a href="https://www.psypost.org/prenatal-cannabis-exposure-linked-to-blunted-brain-response-and-psychotic-like-symptoms-in-youth/" rel="nofollow noopener" translate="no" target="_blank">https://www.psypost.org/prenatal-cannabis-exposure-linked-to-blunted-brain-response-and-psychotic-like-symptoms-in-youth/</a>An analysis of longitudinal data from the Adolescent Brain and Cognitive Development (ABCD) study has found that prenatal exposure to cannabis is associated with an increased likelihood of developing psychotic-like experiences in youth. These experiences were associated with reduced neural activation during reward anticipation in the brain’s reward-processing system, particularly in the striatum—a region involved in motivation and reward expectancy. This blunted response was more pronounced in children who had been exposed to cannabis in utero. The findings were published in Biological Psychiatry.Psychotic-like experiences are subclinical symptoms that resemble features of psychosis but occur in individuals who do not meet the diagnostic criteria for a psychotic disorder. Such experiences may include hearing voices, holding unusual beliefs, experiencing paranoid thoughts, or feeling detached from reality. These symptoms are relatively common during adolescence, with many young people reporting at least one such experience during this developmental period.In most cases, psychotic-like experiences are temporary and do not develop into a clinical disorder. However, frequent or distressing psychotic-like experiences have been linked to increased risk for future psychiatric conditions, including schizophrenia, depression, and anxiety. Factors associated with a heightened risk for psychotic-like experiences include genetic predisposition, childhood trauma, bullying, substance use, and high levels of stress. These experiences may also reflect normative developmental processes as adolescents navigate identity formation and shifts in cognitive and emotional regulation.The study, led by Carolyn M. Amir and Carrie E. Bearden, tested the hypothesis that psychotic-like experiences are associated with diminished neural activity in reward-related brain regions—specifically the striatum and the ventromedial prefrontal cortex—during anticipation of a reward. The researchers also proposed that this association would be stronger in youth who had been exposed to cannabis in utero.“Our lab has been really interested in mechanisms underlying the relationship between cannabis exposure and psychosis in adolescence. Particularly concerning is the rise in cannabis use during pregnancy—and the lack of data on how this might affect brain development,” explained Bearden, a professor and director of the Center for the Assessment and Prevention of Prodromal States (CAPPS) at the University of California, Los Angeles.Prenatal cannabis exposure occurs when a pregnant individual uses cannabis, allowing compounds such as tetrahydrocannabinol (THC) to cross the placenta and affect fetal development. Previous studies have linked this exposure to outcomes such as reduced birth weight, neurodevelopmental alterations, and increased risk for cognitive and behavioral difficulties in childhood.The current study analyzed data from children born between 2005 and 2009 who were enrolled in the ABCD study, a large-scale longitudinal investigation of brain development and health in U.S. youth. The sample included 11,368 participants with an average age of approximately 10 years at baseline.To assess psychotic-like experiences, the researchers used the Prodromal Questionnaire – Brief Child Version, a validated tool designed to measure subclinical psychosis-related symptoms in youth. Information about prenatal cannabis exposure was based on retrospective reports provided by the child’s parent or caregiver. Neural responses were assessed using task-based functional MRI while participants completed the Monetary Incentive Delay task, which measures brain activation during reward anticipation.The results showed that children with prenatal cannabis exposure were more likely to report psychotic-like experiences. Across the entire sample, greater severity of psychotic-like experiences was associated with reduced activation in the striatum during reward anticipation. This effect was significantly stronger among children with prenatal cannabis exposure.The researchers also tested a mediation model to examine whether blunted neural responses to reward could help explain the link between prenatal cannabis exposure and psychotic-like experiences. The findings suggest that reduced striatal activation partially mediated this relationship, indicating that altered reward processing may be one pathway through which prenatal cannabis exposure increases risk for psychotic-like experiences.Additionally, the study found that psychotic-like experiences were associated with higher levels of reward motivation and impulsivity, and that these associations were more pronounced among youth with prenatal cannabis exposure. In this group, participants scored higher on measures of reward drive, reward responsiveness, and multiple dimensions of impulsivity across several assessment time points.“The big finding is that youth who were exposed to cannabis before they were born were more likely to report psychotic-like experiences as teenagers,” Bearden told PsyPost. “This also appeared in how their brains responded to a reward cue — a particular part of the brain, the ventral striatum, that’s normally very reactive in response to anticipating a reward like money, was less activated in children who had been exposed to cannabis prenatally. And that brain response, in turn, was associated with more distressing psychotic-like experiences.”“I was quite surprised that, when controlling for all the possible confounds we could, this association persisted. That exposure during early development would be associated with brain and behavioral differences so many years later was unexpected.”The study sheds light on the links between prenatal cannabis exposure and psychological outcomes later in adolescence. However, it should be noted that the design of this study does not allow any definitive causal inferences to be derived from the results.Looking forward, “we are interested in looking at the relationship of early exposure to both psychotic-like experiences over time, as well as other mental health and substance use outcomes later in life (as more timepoint data from ABCD become available),” Bearden said. “We’re also planning prospective studies with more detailed information on cannabis use—such as potency, frequency, and type of use—and how that relates to both blood biomarkers of cannabinoids as well as symptoms over time.”The paper, “Altered neurobehavioral reward response predicts psychotic-like experiences in youth exposed to cannabis prenatally,” was authored by Carolyn M. Amir, Dara G. Ghahremani, Sarah E. Chang, Ziva D. Cooper, and Carrie E. Bearden.URL: <a href="https://www.psypost.org/prenatal-cannabis-exposure-linked-to-blunted-brain-response-and-psychotic-like-symptoms-in-youth/" rel="nofollow noopener" translate="no" target="_blank">https://www.psypost.org/prenatal-cannabis-exposure-linked-to-blunted-brain-response-and-psychotic-like-symptoms-in-youth/</a>-------------------------------------------------Private, vetted email list for mental health professionals: <a href="https://www.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">https://www.clinicians-exchange.org</a>Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot <a href="https://mastodon.clinicians-exchange.org/@PTUnofficialBot" class="u-url mention" rel="nofollow noopener" target="_blank">@PTUnofficialBot</a>NYU Information for Practice puts out 400-500 good quality health-related research posts per week but its too much for many people, so that bot is limited to just subscribers. 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Military Psychology News RobotTRIGGER WARNING: Military PsychologyDATE: August 22, 2025 at 09:50AM SOURCE: THE CENTER FOR DEPLOYMENT PSYCHOLOGYDirect article link at end of text block below.Any parent would tell you the “why” questions start when we are young, usually around 2- or 3-years-old. The constant need to know the reason behind function, choices, and existence is inherent in us. Read the blog, "Staff Perspective: The Lingering Why". <a href="https://t.co/6AEH6YdEsR" rel="nofollow noopener" translate="no" target="_blank">https://t.co/6AEH6YdEsR</a>Here are any URLs found in the article text: <a href="https://t.co/6AEH6YdEsR" rel="nofollow noopener" translate="no" target="_blank">https://t.co/6AEH6YdEsR</a>Articles can be found by scrolling down the page at <a href="https://deploymentpsych.org/" rel="nofollow noopener" translate="no" target="_blank">https://deploymentpsych.org/</a> under "Latest News". -------------------------------------------------Private, vetted email list for mental health professionals: <a href="https://www.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">https://www.clinicians-exchange.org</a>NYU Information for Practice puts out 400-500 good quality health-related research posts per week but its too much for many people, so that bot is limited to just subscribers. 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Military Psychology News RobotTRIGGER WARNING: Military PsychologyDATE: August 22, 2025 at 09:54AM SOURCE: THE CENTER FOR DEPLOYMENT PSYCHOLOGYDirect article link at end of text block below.Participate in a Content Validity Study of a New Dream Measure- The Research Team at CDP is developing a new approach to assess for nightmares. If you are interested in learning more or participating, please contact us at cat-research.cdp@usuhs.edu to learn more. Articles can be found by scrolling down the page at <a href="https://deploymentpsych.org/" rel="nofollow noopener" translate="no" target="_blank">https://deploymentpsych.org/</a> under "Latest News". -------------------------------------------------Private, vetted email list for mental health professionals: <a href="https://www.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">https://www.clinicians-exchange.org</a>NYU Information for Practice puts out 400-500 good quality health-related research posts per week but its too much for many people, so that bot is limited to just subscribers. You can read it or subscribe at <a href="https://mastodon.clinicians-exchange.org/@PsychResearchBot" class="u-url mention" rel="nofollow noopener" target="_blank">@PsychResearchBot</a>Since 1991 The National Psychologist has focused on keeping practicing psychologists current with news, information and items of interest. 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Psychology News RobotDATE: August 22, 2025 at 09:19AM SOURCE: SCIENCE DAILY MIND-BRAIN FEEDTITLE: A startling omega-3 deficiency may explain women’s Alzheimer’s riskURL: <a href="https://www.sciencedaily.com/releases/2025/08/250821094533.htm" rel="nofollow noopener" translate="no" target="_blank">https://www.sciencedaily.com/releases/2025/08/250821094533.htm</a>Researchers discovered that women with Alzheimer’s show a sharp loss of omega fatty acids, unlike men, pointing to sex-specific differences in the disease. The study suggests omega-rich diets could be key, but clinical trials are needed.URL: <a href="https://www.sciencedaily.com/releases/2025/08/250821094533.htm" rel="nofollow noopener" translate="no" target="_blank">https://www.sciencedaily.com/releases/2025/08/250821094533.htm</a>-------------------------------------------------Private, vetted email list for mental health professionals: <a href="https://www.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">https://www.clinicians-exchange.org</a>Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot <a href="https://mastodon.clinicians-exchange.org/@PTUnofficialBot" class="u-url mention" rel="nofollow noopener" target="_blank">@PTUnofficialBot</a>NYU Information for Practice puts out 400-500 good quality health-related research posts per week but its too much for many people, so that bot is limited to just subscribers. You can read it or subscribe at <a href="https://mastodon.clinicians-exchange.org/@PsychResearchBot" class="u-url mention" rel="nofollow noopener" target="_blank">@PsychResearchBot</a>Since 1991 The National Psychologist has focused on keeping practicing psychologists current with news, information and items of interest. Check them out for more free articles, resources, and subscription information: <a href="https://www.nationalpsychologist.com" rel="nofollow noopener" translate="no" target="_blank">https://www.nationalpsychologist.com</a>EMAIL DAILY DIGEST OF RSS FEEDS -- SUBSCRIBE: <a href="http://subscribe-article-digests.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">http://subscribe-article-digests.clinicians-exchange.org</a>READ ONLINE: <a href="http://read-the-rss-mega-archive.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">http://read-the-rss-mega-archive.clinicians-exchange.org</a>It's primitive... but it works... mostly...-------------------------------------------------<a href="https://mastodon.clinicians-exchange.org/tags/psychology" class="mention hashtag" rel="nofollow noopener" target="_blank">#psychology</a> <a href="https://mastodon.clinicians-exchange.org/tags/counseling" class="mention hashtag" rel="nofollow noopener" target="_blank">#counseling</a> <a href="https://mastodon.clinicians-exchange.org/tags/socialwork" class="mention hashtag" rel="nofollow noopener" target="_blank">#socialwork</a> <a href="https://mastodon.clinicians-exchange.org/tags/psychotherapy" class="mention hashtag" rel="nofollow noopener" target="_blank">#psychotherapy</a> <a href="https://a.gup.pe/u/psychotherapist" class="u-url mention" rel="nofollow noopener" target="_blank">@psychotherapist</a> <a href="https://a.gup.pe/u/psychotherapists" class="u-url mention" rel="nofollow noopener" target="_blank">@psychotherapists</a> <a href="https://a.gup.pe/u/psychology" class="u-url mention" rel="nofollow noopener" target="_blank">@psychology</a> <a href="https://a.gup.pe/u/socialpsych" class="u-url mention" rel="nofollow noopener" target="_blank">@socialpsych</a> <a href="https://a.gup.pe/u/socialwork" class="u-url mention" rel="nofollow noopener" target="_blank">@socialwork</a> <a href="https://a.gup.pe/u/psychiatry" class="u-url mention" rel="nofollow noopener" target="_blank">@psychiatry</a> <a href="https://mastodon.clinicians-exchange.org/tags/mentalhealth" class="mention hashtag" rel="nofollow noopener" target="_blank">#mentalhealth</a> <a href="https://mastodon.clinicians-exchange.org/tags/psychiatry" class="mention hashtag" rel="nofollow noopener" target="_blank">#psychiatry</a> <a href="https://mastodon.clinicians-exchange.org/tags/healthcare" class="mention hashtag" rel="nofollow noopener" target="_blank">#healthcare</a> <a href="https://mastodon.clinicians-exchange.org/tags/depression" class="mention hashtag" rel="nofollow noopener" target="_blank">#depression</a> <a href="https://mastodon.clinicians-exchange.org/tags/psychotherapist" class="mention hashtag" rel="nofollow noopener" target="_blank">#psychotherapist</a>

Psychology News RobotDATE: August 22, 2025 at 09:00AM SOURCE: PSYCHIATRIC TIMESDirect article link at end of text block below.Antidepressant treatment for late-life depression may improve cognitive domains such as memory, learning, and processing speed, with improvements potentially linked to reductions in depressive symptoms. <a href="https://t.co/xbHgXdSdF3" rel="nofollow noopener" translate="no" target="_blank">https://t.co/xbHgXdSdF3</a>Here are any URLs found in the article text: <a href="https://t.co/xbHgXdSdF3" rel="nofollow noopener" translate="no" target="_blank">https://t.co/xbHgXdSdF3</a>Articles can be found by scrolling down the page at Articles can be found at <a href="https://www.psychiatrictimes.com/news" rel="nofollow noopener" translate="no" target="_blank">https://www.psychiatrictimes.com/news</a>". -------------------------------------------------Private, vetted email list for mental health professionals: <a href="https://www.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">https://www.clinicians-exchange.org</a>NYU Information for Practice puts out 400-500 good quality health-related research posts per week but its too much for many people, so that bot is limited to just subscribers. You can read it or subscribe at <a href="https://mastodon.clinicians-exchange.org/@PsychResearchBot" class="u-url mention" rel="nofollow noopener" target="_blank">@PsychResearchBot</a>-------------------------------------------------<a href="https://mastodon.clinicians-exchange.org/tags/psychology" class="mention hashtag" rel="nofollow noopener" target="_blank">#psychology</a> <a href="https://mastodon.clinicians-exchange.org/tags/counseling" class="mention hashtag" rel="nofollow noopener" target="_blank">#counseling</a> <a href="https://mastodon.clinicians-exchange.org/tags/socialwork" class="mention hashtag" rel="nofollow noopener" target="_blank">#socialwork</a> <a href="https://mastodon.clinicians-exchange.org/tags/psychotherapy" class="mention hashtag" rel="nofollow noopener" target="_blank">#psychotherapy</a> <a href="https://a.gup.pe/u/psychotherapist" class="u-url mention" rel="nofollow noopener" target="_blank">@psychotherapist</a> <a href="https://a.gup.pe/u/psychotherapists" class="u-url mention" rel="nofollow noopener" target="_blank">@psychotherapists</a> <a href="https://a.gup.pe/u/psychology" class="u-url mention" rel="nofollow noopener" target="_blank">@psychology</a> <a href="https://a.gup.pe/u/socialpsych" class="u-url mention" rel="nofollow noopener" target="_blank">@socialpsych</a> <a href="https://a.gup.pe/u/socialwork" class="u-url mention" rel="nofollow noopener" target="_blank">@socialwork</a> <a href="https://a.gup.pe/u/psychiatry" class="u-url mention" rel="nofollow noopener" target="_blank">@psychiatry</a> <a href="https://mastodon.clinicians-exchange.org/tags/mentalhealth" class="mention hashtag" rel="nofollow noopener" target="_blank">#mentalhealth</a> <a href="https://mastodon.clinicians-exchange.org/tags/psychiatry" class="mention hashtag" rel="nofollow noopener" target="_blank">#psychiatry</a> <a href="https://mastodon.clinicians-exchange.org/tags/healthcare" class="mention hashtag" rel="nofollow noopener" target="_blank">#healthcare</a> <a href="https://mastodon.clinicians-exchange.org/tags/psychotherapist" class="mention hashtag" rel="nofollow noopener" target="_blank">#psychotherapist</a>

Healthcare Tech RobotDATE: August 22, 2025 at 08:30AM SOURCE: DIGITALHEALTH.NETTITLE: Cancer Awareness Trust to pilot support platform in the UKURL: <a href="https://www.digitalhealth.net/2025/08/cancer-awareness-trust-to-pilot-support-platform-in-the-uk/" rel="nofollow noopener" translate="no" target="_blank">https://www.digitalhealth.net/2025/08/cancer-awareness-trust-to-pilot-support-platform-in-the-uk/</a>Cancer Awareness Trust is launching a platform in the UK to support people affected by cancer, designed in partnership with ustwo.URL: <a href="https://www.digitalhealth.net/2025/08/cancer-awareness-trust-to-pilot-support-platform-in-the-uk/" rel="nofollow noopener" translate="no" target="_blank">https://www.digitalhealth.net/2025/08/cancer-awareness-trust-to-pilot-support-platform-in-the-uk/</a>-------------------------------------------------Private, vetted email list for mental health professionals: <a href="https://www.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">https://www.clinicians-exchange.org</a> . NYU Information for Practice puts out 400-500 good quality health-related research posts per week but its too much for many people, so that bot is limited to just subscribers. You can read it or subscribe at <a href="https://mastodon.clinicians-exchange.org/@PsychResearchBot" class="u-url mention" rel="nofollow noopener" target="_blank">@PsychResearchBot</a> . Since 1991 The National Psychologist has focused on keeping practicing psychologists current with news, information and items of interest. Check them out for more free articles, resources, and subscription information: <a href="https://www.nationalpsychologist.com" rel="nofollow noopener" translate="no" target="_blank">https://www.nationalpsychologist.com</a> . EMAIL DAILY DIGEST OF RSS FEEDS -- SUBSCRIBE: <a href="http://subscribe-article-digests.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">http://subscribe-article-digests.clinicians-exchange.org</a> . 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Psychology News RobotDATE: August 22, 2025 at 08:00AM SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. ** -------------------------------------------------TITLE: Scientist who linked autism to chemical and pharmaceutical exposures saw her entire division shut down by RFK Jr.URL: <a href="https://www.psypost.org/scientist-who-linked-autism-to-chemical-and-pharmaceutical-exposures-saw-her-entire-division-shut-down-by-rfk-jr/" rel="nofollow noopener" translate="no" target="_blank">https://www.psypost.org/scientist-who-linked-autism-to-chemical-and-pharmaceutical-exposures-saw-her-entire-division-shut-down-by-rfk-jr/</a>A large new study published in the International Journal of Hygiene and Environmental Health provides evidence that exposure to certain workplace chemicals among parents may influence the severity of autism spectrum disorder (ASD) symptoms and contribute to behavioral, cognitive, and adaptive challenges in their children. The findings suggest that occupational exposures—especially to plastics, ethylene oxide, phenols, and pharmaceutical agents—may have broader developmental effects beyond autism diagnosis alone.Autism spectrum disorder is typically understood as a neurodevelopmental condition involving a mix of genetic and environmental factors. While previous research has associated prenatal exposure to environmental pollutants with increased autism risk, most of that work has focused on diagnosis itself, not on the range or severity of symptoms children may later experience. This study sought to expand that understanding by examining whether parental occupational exposures to potentially harmful agents before and during pregnancy are associated with the intensity of ASD symptoms and co-occurring developmental difficulties.The study was conducted by former researchers from the CDC’s National Institute for Occupational Safety and Health (NIOSH), in collaboration with the University of California, Davis, as part of the CHARGE (Childhood Autism Risks from Genetics and Environment) study. Their aim was to test whether parents’ workplace exposure to chemicals might contribute not just to ASD risk but to more severe impairments in communication, cognition, or adaptive functioning in children who are already diagnosed.“My stepson was working with children with autism and we began discussing the spectrum of symptoms that children with autism experience and that little was understood about the causes of autism,” explained study author Erin C. McCanlies.McCanlies had worked at NIOSH’s Health Effects Laboratory Division as a research epidemiologist, where she studied the impact of occupational exposures on health. That division was responsible for conducting research on workplace health and safety. She is now retired after the division was eliminated under the direction of Health and Human Services Secretary Robert F. Kennedy Jr.“My job was considering how occupational exposures may result in illness or injury. This led to my thinking about the possible causes behind autism, particularly chemicals and other agents that parents might be exposed to while on the job that could potentially result in autism in their children. To better understand this potential risk I reached out to Dr. Irva Hertz-Picciotto at UC Davis, who was conducting a large study, the CHARGE study, which was designed to evaluate genetics and environmental factors that might be involved in the causes of autism.”The researchers analyzed data from 532 children with confirmed ASD diagnoses and at least one parent with complete occupational history. All children were between the ages of 2 and 5 and participated in the CHARGE study, a large case-control investigation of autism and developmental delays in California. Each child’s ASD diagnosis was validated using gold-standard instruments such as the Autism Diagnostic Observation Schedule (ADOS-2) and the Autism Diagnostic Interview-Revised (ADI-R).Parents completed extensive surveys about their work histories, including the types of jobs they held and any potential exposures to harmful substances during the “index period”—defined as the three months before conception through the end of pregnancy. Two industrial hygienists reviewed each job entry and assigned exposure ratings to a panel of 16 chemical categories, including disinfectants, solvents, metals, plastics, phenols, and others. These ratings were combined into a cumulative exposure score for each parent.To assess ASD symptom severity, the researchers used a standardized metric called the Calibrated Severity Score (CSS), derived from ADOS-2 results. This score is intended to capture core autism features independently of age or cognitive ability.In addition to the CSS, they evaluated cognitive function using the Mullen Scales of Early Learning (MSEL), adaptive functioning with the Vineland Adaptive Behavior Scales (VABS), and problem behaviors with the Aberrant Behavior Checklist (ABC). These instruments provided a broader picture of each child’s developmental profile, allowing the team to examine how parental exposures might be linked to different aspects of a child’s functioning.The findings suggest that workplace exposures to several specific chemical classes were associated with worse outcomes in children with ASD. One of the strongest and most consistent patterns involved plastics and polymer chemicals. Fathers’ exposure to plastics was associated with lower scores across all cognitive and adaptive skill domains, including language, motor coordination, daily living skills, and overall functioning. When both parents were exposed, the deficits appeared to compound.“I was surprised how strongly and consistently plastics and polymers stood out as being linked with multiple developmental and behavioral outcomes including irritability, hyperactivity, and daily living,” McCanlies told PsyPost.Exposure to ethylene oxide—commonly used in hospital sterilization—was also linked to more severe autism symptoms, lower expressive language abilities, and poorer adaptive functioning. Similarly, parental exposure to phenol (used in construction, automotive, and some consumer products) and pharmaceuticals was associated with increased ASD severity and more pronounced behavioral challenges, especially hyperactivity and stereotyped behavior.While the results do not imply that all children exposed to these chemicals will develop more severe symptoms, the patterns suggest that early life exposure to workplace toxicants may amplify certain developmental difficulties in children who already meet criteria for ASD. The study provides one of the most detailed looks to date at how parental occupation may relate not just to diagnosis, but to variation in how autism is expressed.“Our findings suggest that certain parental workplace exposures may be related not just to autism, but to worse symptoms and autism behaviors,” McCanlies explained.These findings are consistent with past research linking prenatal exposure to chemicals like BPA, phthalates, and metals to developmental delays, behavioral issues, and increased autism risk. The current study builds on that work by identifying specific occupational chemicals that may intensify these outcomes.“These findings suggest that both men and women should follow recommended safety practices and use protective gear when working with these chemicals,” McCanlies said.“This research shows that workplace safety isn’t just about protecting the worker — it’s also about protecting their future children,” added Hertz-Picciotto, a professor at the UC Davis MIND Institute. “We must consider how workplace chemicals might affect the next generation.”The researchers suggest several possible biological mechanisms that could explain these associations. Many of the chemicals studied are known endocrine disruptors or immune-modulating agents, which can interfere with hormonal signaling, neural development, or inflammatory processes during pregnancy. Micro- and nano-plastics, in particular, have been shown in animal studies to cross the placenta and disrupt gene expression in the developing brain.While the study offers strong statistical support for several associations, it is observational in nature and cannot prove causation. Exposure estimates were based on job descriptions rather than direct biological measurements, which may lead to some misclassification. In cases where mothers reported fathers’ job histories, the accuracy of those reports may have varied. Additionally, some chemicals had too few exposures to draw firm conclusions.“More research is needed to better understand how these exposures affect brain development,” McCanlies noted. “Although we had a fairly large number of families, the number was still too small to see links with less common exposures.”Nonetheless, the use of validated diagnostic tools, a large sample, expert-reviewed exposure ratings, and multiple outcome measures adds weight to the findings. The researchers note that misclassification errors in this study would most likely bias results toward the null, meaning the observed associations may underrepresent the true effects.“Next steps would include longitudinal studies investigating workplace environments and specific exposures that affect both mothers and fathers that may be associated with autism and its severity,” McCanlies explained. “Research aimed at understanding the biological mechanisms underlying the link between work exposures and autism and its severity will also be important to confirm plausibility of causation and for developing effective intervention and prevention methods.”“This study is unique; there are few studies that have evaluated parent work exposures and autism severity and behaviors,” McCanlies added.The study, “The effects of parental occupational exposures on autism spectrum disorder severity and skills in cognitive and adaptive domains in children with autism spectrum disorder,” was authored by Erin C. 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Ludeña-Rodriguez, and Irva Hertz-Picciotto.URL: <a href="https://www.psypost.org/scientist-who-linked-autism-to-chemical-and-pharmaceutical-exposures-saw-her-entire-division-shut-down-by-rfk-jr/" rel="nofollow noopener" translate="no" target="_blank">https://www.psypost.org/scientist-who-linked-autism-to-chemical-and-pharmaceutical-exposures-saw-her-entire-division-shut-down-by-rfk-jr/</a>-------------------------------------------------Private, vetted email list for mental health professionals: <a href="https://www.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">https://www.clinicians-exchange.org</a>Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot <a href="https://mastodon.clinicians-exchange.org/@PTUnofficialBot" class="u-url mention" rel="nofollow noopener" target="_blank">@PTUnofficialBot</a>NYU Information for Practice puts out 400-500 good quality health-related research posts per week but its too much for many people, so that bot is limited to just subscribers. 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Healthcare Tech RobotDATE: August 22, 2025 at 06:00AM SOURCE: DIGITALHEALTH.NETTITLE: Torbay Council to roll out System C software for adult social careURL: <a href="https://www.digitalhealth.net/2025/08/torbay-council-to-roll-out-system-c-software-for-adult-social-care/" rel="nofollow noopener" translate="no" target="_blank">https://www.digitalhealth.net/2025/08/torbay-council-to-roll-out-system-c-software-for-adult-social-care/</a>Torbay Council has extended its partnership with System C to implement its adult social care case management and finance system.URL: <a href="https://www.digitalhealth.net/2025/08/torbay-council-to-roll-out-system-c-software-for-adult-social-care/" rel="nofollow noopener" translate="no" target="_blank">https://www.digitalhealth.net/2025/08/torbay-council-to-roll-out-system-c-software-for-adult-social-care/</a>-------------------------------------------------Private, vetted email list for mental health professionals: <a href="https://www.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">https://www.clinicians-exchange.org</a> . 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Psychology News RobotDATE: August 22, 2025 at 06:00AM SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. ** -------------------------------------------------TITLE: Antidepressant withdrawal symptoms may be more common and more severe than some studies suggestURL: <a href="https://www.psypost.org/antidepressant-withdrawal-symptoms-may-be-more-common-and-more-severe-than-some-studies-suggest/" rel="nofollow noopener" translate="no" target="_blank">https://www.psypost.org/antidepressant-withdrawal-symptoms-may-be-more-common-and-more-severe-than-some-studies-suggest/</a>A newly published study in Psychological Medicine raises questions about a widely cited 2024 meta-analysis that downplayed the frequency and severity of antidepressant withdrawal symptoms. The new reanalysis suggests that when studies use systematic and appropriate methods to assess withdrawal effects, more than half of patients report experiencing symptoms—contradicting earlier claims that only about 15% are affected. The findings challenge current perceptions about the risks associated with stopping antidepressant use, particularly after short-term treatment.Antidepressants are among the most commonly prescribed psychiatric medications in high-income countries, and many patients remain on them for extended periods. Yet questions remain about what happens when people stop taking them.In 2024, a large meta-analysis published in The Lancet Psychiatry sought to clarify just how often patients experience discontinuation symptoms after stopping antidepressants. The authors of that review, including Jonathan Henssler and colleagues based in Germany, aimed to produce what they described as a more comprehensive estimate of the incidence and severity of withdrawal effects.Their study found that about 15% of people who stop taking antidepressants experience one or more withdrawal symptoms that can be directly attributed to the medication. These include issues such as dizziness, nausea, insomnia, and irritability. The researchers also reported that only about 3% of patients experienced what they considered severe symptoms—those intense enough to lead to dropout from a study or the resumption of antidepressant treatment.To conduct the analysis, Henssler and colleagues systematically reviewed data from 79 studies published between 1961 and 2019, encompassing more than 21,000 participants. Their inclusion criteria were broad: the team accepted both randomized and non-randomized trials, as well as observational studies, as long as they assessed withdrawal symptoms following the discontinuation of antidepressants or placebo. They also analyzed withdrawal symptoms reported after stopping placebos in controlled trials, which they used as a baseline to subtract non-specific symptoms that might be misattributed to withdrawal.Of the participants, more than 16,000 had discontinued antidepressant treatment, while nearly 4,500 had discontinued placebo. Across all groups, the researchers estimated that roughly 31% of patients experienced some form of withdrawal symptom after stopping antidepressants.After accounting for the incidence of symptoms in placebo groups—estimated at about 17%—the authors concluded that roughly 15% of the symptoms could be directly tied to antidepressant withdrawal. They also identified some antidepressants, such as imipramine, paroxetine, and venlafaxine, as carrying a relatively higher risk of withdrawal symptoms.The study attracted attention for its size and its conclusion that withdrawal effects, while real, were not particularly frequent or severe. But critics soon raised concerns about how the data had been collected and analyzed. In particular, several researchers questioned whether the methods used in many of the included studies were sufficient to detect withdrawal symptoms accurately.That skepticism prompted the publication of a new study in Psychological Medicine, led by Joanna Moncrieff of University College London. Along with a team of researchers from Denmark, Switzerland, Austria, and the United Kingdom, Moncrieff sought to examine the data used in the original review and determine whether its conclusions were supported by more rigorous assessments.“We did this research because we are concerned that some psychiatrists tend to minimize the adverse effects associated with antidepressants, particularly their dependence-inducing effects,” explained Moncrieff, a professor of critical and social psychiatry and author of Chemically Imbalanced. “This may be because psychiatrists see the medical treatment of depression as central to the enterprise of psychiatry, and therefore feel the need to defend the reputation of antidepressants come what may.”To assess the reliability of the original review, Moncrieff and her colleagues reviewed all 62 study cohorts that Henssler’s team had used to estimate the incidence of withdrawal symptoms. They examined whether those studies had used systematic and specific methods to measure symptoms, or whether they had relied on more casual observations—such as spontaneous adverse event reporting or non-specific clinician notes.The researchers found that only five of the 62 studies—less than 10%—had measured withdrawal symptoms using structured questionnaires or tools specifically designed to assess the kinds of symptoms typically reported during antidepressant discontinuation. These tools, such as the Discontinuation-Emergent Signs and Symptoms (DESS) checklist, capture a range of common experiences including dizziness, emotional instability, and “brain zaps.” In contrast, most of the studies relied on vague or inconsistent methods, including open-ended questions, clinician impressions, or even retrospective chart reviews.“The majority of studies were not proper studies of withdrawal but had looked at withdrawal only casually and incidentally,” Moncrieff told PsyPost. “In fact, very few of the studies had used a proper measure of withdrawal symptoms.”The new team also raised concerns about other methodological features of the included studies. Many of the trials were short in duration, sometimes observing patients for only two weeks after discontinuation—a period that may not capture delayed withdrawal symptoms. The average length of antidepressant use before stopping was also less than six months in most studies, which could reduce the likelihood of detecting symptoms that become more likely with long-term use.Another issue involved the way that withdrawal symptoms were sometimes misclassified as a return of the underlying mental health condition, especially in studies where symptoms of depression or anxiety were being monitored alongside withdrawal effects. This made it harder to distinguish between a relapse of the original disorder and genuine discontinuation symptoms.The researchers then conducted their own meta-analysis, including only those five studies that had systematically measured withdrawal symptoms. These studies involved 601 participants, most of whom had used antidepressants for three months or less. They found that 55% of participants experienced at least one withdrawal symptom after stopping their medication. When they excluded one study in which patients did not fully stop their medication, the estimate rose to 61%. Excluding a trial of agomelatine, which is known to have lower dependence potential, pushed the estimate to 69%.“We found that of the 62 studies included in the original review, only 5 used a systematic measure of antidepressant withdrawal symptoms,” Moncrieff explained. “Four of these involved people who had only used antidepressants short-term (up to 12 weeks). When we looked at the findings of these studies specifically, between 55% and 69% of participants reported experiencing withdrawal symptoms after stopping their antidepressant.”“So overall, we found that the majority of evidence on antidepressant withdrawal is of poor quality and does not involve people who have taken antidepressants for long periods (those who are most at risk). The evidence that exists, however, suggests antidepressants may induce withdrawal reactions quite frequently, even after short-term use.”These findings suggest that when withdrawal is measured systematically, a much larger proportion of patients report symptoms than the earlier review had indicated. The authors emphasize that their estimates do not subtract rates of withdrawal-like symptoms in placebo groups, but argue that the types and severity of symptoms reported after antidepressant discontinuation are not equivalent to those seen with placebo.The new study concludes that the original 2024 review underestimates the incidence and severity of antidepressant withdrawal, in large part because of limitations in the data it relied on. These included inadequate assessment methods, short follow-up periods, and the frequent misclassification of symptoms. The researchers argue that better-designed studies are needed to understand the full scope of the problem—particularly for individuals who have used antidepressants for longer than a few months.While both reviews agree that withdrawal symptoms do occur, they offer very different pictures of how often they happen and how disruptive they might be. One suggests that withdrawal is relatively rare and mild; the other indicates that it may be common, even after short-term use, and that current clinical guidelines may not reflect the full reality experienced by patients.Another recent study, published in JAMA Psychiatry, which involved a meta-analysis of 49 randomized clinical trials, found that people who stop taking antidepressants tend to experience only one additional discontinuation symptom, on average, compared to those who discontinue placebo or remain on the medication. The most common symptom within the first two weeks was dizziness, but the overall number and intensity of symptoms remained below the threshold typically used to define clinically significant withdrawal, and no increase in depressive symptoms was observed.However, this newer study has also faced critics, who argue that it downplayed the risks of antidepressant withdrawal by relying on short-term, industry-funded studies, failing to adequately account for the more severe and long-lasting symptoms experienced by long-term users and potentially repeating past mistakes that delayed recognition of the issue.“Another review of antidepressant withdrawal was published recently, which also used short-term trials and poor data to come to the conclusion that antidepressant withdrawal is not a significant problem,” Moncrieff said. “However, in fact, the studies included in this latest review revealed that antidepressant withdrawal does exist, even after short-term use.”“Evidence from surveys and thousands of patient reports points to the fact that antidepressant withdrawal is a significant clinical problem. Attempts to minimize it are worrying because it means that the many people who suffer from severe withdrawal reactions will not receive the understanding and support they need.”As the debate continues, the need for high-quality, independent research remains urgent—particularly studies that measure withdrawal with the same level of care used to evaluate drug efficacy.“There is little good quality data on antidepressant withdrawal, so it is difficult to make estimates about its frequency,” Moncrieff told PsyPost. “Also, it likely that some antidepressants have stronger dependence-inducing effects than others. Withdrawal symptoms include common symptoms such as headaches, dizziness, anxiety, and other complaints, and so far there are no studies that try to distinguish between withdrawal symptoms and ‘background’ symptoms. I hope to set up a study that can explore antidepressant withdrawal in more detail and also how to help people avoid severe withdrawal symptoms.”The new study, “Evidence on antidepressant withdrawal: an appraisal and reanalysis of a recent systematic review,” was authored by Joanna Moncrieff, Harriet Hobday, Anders Sørensen, John Read, Martin Plöderl, Michael Hengartner, Caroline Kamp, Janus Jakobsen, Sophie Juul, James Davies and Mark Horowitz.URL: <a href="https://www.psypost.org/antidepressant-withdrawal-symptoms-may-be-more-common-and-more-severe-than-some-studies-suggest/" rel="nofollow noopener" translate="no" target="_blank">https://www.psypost.org/antidepressant-withdrawal-symptoms-may-be-more-common-and-more-severe-than-some-studies-suggest/</a>-------------------------------------------------Private, vetted email list for mental health professionals: <a href="https://www.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">https://www.clinicians-exchange.org</a>Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot <a href="https://mastodon.clinicians-exchange.org/@PTUnofficialBot" class="u-url mention" rel="nofollow noopener" target="_blank">@PTUnofficialBot</a>NYU Information for Practice puts out 400-500 good quality health-related research posts per week but its too much for many people, so that bot is limited to just subscribers. 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Psychology News RobotDATE: August 22, 2025 at 04:30AM SOURCE: STAT NEWS MENTAL HEALTHTITLE: Opinion: Hospitals should not separate postpartum women in crisis from their newbornsURL: <a href="https://www.statnews.com/2025/08/22/postpartum-depression-hospital-emergency-room-separate-mothers-babies/?utm_campaign=rss" rel="nofollow noopener" translate="no" target="_blank">https://www.statnews.com/2025/08/22/postpartum-depression-hospital-emergency-room-separate-mothers-babies/?utm_campaign=rss</a>A woman came into triage with her 6-week-old baby, terrified. She had little support at home and was worried about caring for her new baby alone. Exhausted, she feared she might harm herself, and so she did what she had been told to do: She called her doctor.Up to 1 in 5 women suffer from mental health or substance use disorders during pregnancy or in the year after they give birth. However, many do not seek help. This patient did. Of course, her doctor was concerned and recommended she go to the emergency room. This was the only suggestion the doctor had been trained to give for what to tell patients in a crisis like this.Read the rest…URL: <a href="https://www.statnews.com/2025/08/22/postpartum-depression-hospital-emergency-room-separate-mothers-babies/?utm_campaign=rss" rel="nofollow noopener" translate="no" target="_blank">https://www.statnews.com/2025/08/22/postpartum-depression-hospital-emergency-room-separate-mothers-babies/?utm_campaign=rss</a>-------------------------------------------------STAT News reports "from the frontiers of health and medicine".Learn more at <a href="https://www.statnews.com/topic/mental-health/" rel="nofollow noopener" translate="no" target="_blank">https://www.statnews.com/topic/mental-health/</a> .See also their complete Mastodon account at <a href="https://newsie.social/@STAT" class="u-url mention" rel="nofollow noopener" target="_blank">@STAT</a> .This robot is NOT affiliated with STAT news and merely rebroadcasts from their site. Responses posted here are not monitored.-------------------------------------------------Private, vetted email list for mental health professionals: <a href="https://www.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">https://www.clinicians-exchange.org</a>Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot <a href="https://mastodon.clinicians-exchange.org/@PTUnofficialBot" class="u-url mention" rel="nofollow noopener" target="_blank">@PTUnofficialBot</a>NYU Information for Practice puts out 400-500 good quality health-related research posts per week but its too much for many people, so that bot is limited to just subscribers. You can read it or subscribe at <a href="https://mastodon.clinicians-exchange.org/@PsychResearchBot" class="u-url mention" rel="nofollow noopener" target="_blank">@PsychResearchBot</a>Since 1991 The National Psychologist has focused on keeping practicing psychologists current with news, information and items of interest. Check them out for more free articles, resources, and subscription information: <a href="https://www.nationalpsychologist.com" rel="nofollow noopener" translate="no" target="_blank">https://www.nationalpsychologist.com</a>EMAIL DAILY DIGEST OF RSS FEEDS -- SUBSCRIBE: <a href="http://subscribe-article-digests.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">http://subscribe-article-digests.clinicians-exchange.org</a>READ ONLINE: <a href="http://read-the-rss-mega-archive.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">http://read-the-rss-mega-archive.clinicians-exchange.org</a>It's primitive... but it works... mostly...-------------------------------------------------<a href="https://mastodon.clinicians-exchange.org/tags/psychology" class="mention hashtag" rel="nofollow noopener" target="_blank">#psychology</a> <a href="https://mastodon.clinicians-exchange.org/tags/counseling" class="mention hashtag" rel="nofollow noopener" target="_blank">#counseling</a> <a href="https://mastodon.clinicians-exchange.org/tags/socialwork" class="mention hashtag" rel="nofollow noopener" target="_blank">#socialwork</a> <a href="https://mastodon.clinicians-exchange.org/tags/psychotherapy" class="mention hashtag" rel="nofollow noopener" target="_blank">#psychotherapy</a> <a href="https://a.gup.pe/u/psychotherapist" class="u-url mention" rel="nofollow noopener" target="_blank">@psychotherapist</a> <a href="https://a.gup.pe/u/psychotherapists" class="u-url mention" rel="nofollow noopener" target="_blank">@psychotherapists</a> <a href="https://a.gup.pe/u/psychology" class="u-url mention" rel="nofollow noopener" target="_blank">@psychology</a> <a href="https://a.gup.pe/u/socialpsych" class="u-url mention" rel="nofollow noopener" target="_blank">@socialpsych</a> <a href="https://a.gup.pe/u/socialwork" class="u-url mention" rel="nofollow noopener" target="_blank">@socialwork</a> <a href="https://a.gup.pe/u/psychiatry" class="u-url mention" rel="nofollow noopener" target="_blank">@psychiatry</a> <a href="https://mastodon.clinicians-exchange.org/tags/mentalhealth" class="mention hashtag" rel="nofollow noopener" target="_blank">#mentalhealth</a> <a href="https://mastodon.clinicians-exchange.org/tags/psychiatry" class="mention hashtag" rel="nofollow noopener" target="_blank">#psychiatry</a> <a href="https://mastodon.clinicians-exchange.org/tags/healthcare" class="mention hashtag" rel="nofollow noopener" target="_blank">#healthcare</a> <a href="https://mastodon.clinicians-exchange.org/tags/depression" class="mention hashtag" rel="nofollow noopener" target="_blank">#depression</a> <a href="https://mastodon.clinicians-exchange.org/tags/psychotherapist" class="mention hashtag" rel="nofollow noopener" target="_blank">#psychotherapist</a>

Healthcare Tech RobotDATE: August 22, 2025 at 03:30AM SOURCE: DIGITALHEALTH.NETTITLE: Movers and Shakers news roundupURL: <a href="https://www.digitalhealth.net/2025/08/movers-and-shakers-news-roundup-62/" rel="nofollow noopener" translate="no" target="_blank">https://www.digitalhealth.net/2025/08/movers-and-shakers-news-roundup-62/</a>This roundup includes the planned departures of both the chief executive and deputy chief executive at Leeds Teaching Hospitals NHS Trust.URL: <a href="https://www.digitalhealth.net/2025/08/movers-and-shakers-news-roundup-62/" rel="nofollow noopener" translate="no" target="_blank">https://www.digitalhealth.net/2025/08/movers-and-shakers-news-roundup-62/</a>-------------------------------------------------Private, vetted email list for mental health professionals: <a href="https://www.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">https://www.clinicians-exchange.org</a> . NYU Information for Practice puts out 400-500 good quality health-related research posts per week but its too much for many people, so that bot is limited to just subscribers. You can read it or subscribe at <a href="https://mastodon.clinicians-exchange.org/@PsychResearchBot" class="u-url mention" rel="nofollow noopener" target="_blank">@PsychResearchBot</a> . Since 1991 The National Psychologist has focused on keeping practicing psychologists current with news, information and items of interest. Check them out for more free articles, resources, and subscription information: <a href="https://www.nationalpsychologist.com" rel="nofollow noopener" translate="no" target="_blank">https://www.nationalpsychologist.com</a> . EMAIL DAILY DIGEST OF RSS FEEDS -- SUBSCRIBE: <a href="http://subscribe-article-digests.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">http://subscribe-article-digests.clinicians-exchange.org</a> . 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Psychology News RobotDATE: August 22, 2025 at 01:43PM SOURCE: SOCIALPSYCHOLOGY.ORGTITLE: Supreme Court Clears Way for Canceling NIH Grants Tied to GenderURL: <a href="http://www.socialpsychology.org/client/redirect.php?from=rss_feed&id=187074&url=https://www.cbsnews.com/news/supreme-court-clears-trump-admin-cancel-nih-diversity-gender-identity-related-grants/" rel="nofollow noopener" translate="no" target="_blank">http://www.socialpsychology.org/client/redirect.php?from=rss_feed&id=187074&url=https://www.cbsnews.com/news/supreme-court-clears-trump-admin-cancel-nih-diversity-gender-identity-related-grants/</a>Source: CBS News - U.S. NewsIn a 5-4 decision, the U.S. Supreme Court on Thursday cleared the way for the Trump administration to proceed with the cancellation of National Institutes of Health research grants tied to issues like gender identity and diversity, equity, and inclusion. Since returning to the White House, President Trump has directed federal agencies to cancel DEI-related grants or contracts and ensure federal funds do not go toward initiatives involving gender...URL: <a href="http://www.socialpsychology.org/client/redirect.php?from=rss_feed&id=187074&url=https://www.cbsnews.com/news/supreme-court-clears-trump-admin-cancel-nih-diversity-gender-identity-related-grants/" rel="nofollow noopener" translate="no" target="_blank">http://www.socialpsychology.org/client/redirect.php?from=rss_feed&id=187074&url=https://www.cbsnews.com/news/supreme-court-clears-trump-admin-cancel-nih-diversity-gender-identity-related-grants/</a>-------------------------------------------------Private, vetted email list for mental health professionals: <a href="https://www.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">https://www.clinicians-exchange.org</a>Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot <a href="https://mastodon.clinicians-exchange.org/@PTUnofficialBot" class="u-url mention" rel="nofollow noopener" target="_blank">@PTUnofficialBot</a>NYU Information for Practice puts out 400-500 good quality health-related research posts per week but its too much for many people, so that bot is limited to just subscribers. You can read it or subscribe at <a href="https://mastodon.clinicians-exchange.org/@PsychResearchBot" class="u-url mention" rel="nofollow noopener" target="_blank">@PsychResearchBot</a>Since 1991 The National Psychologist has focused on keeping practicing psychologists current with news, information and items of interest. 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Psychology News RobotDATE: August 22, 2025 at 01:43PM SOURCE: SOCIALPSYCHOLOGY.ORGTITLE: Hundreds of Thousands of Grok Chats Exposed in Google ResultsURL: <a href="http://www.socialpsychology.org/client/redirect.php?from=rss_feed&id=187067&url=https://www.bbc.com/news/articles/cdrkmk00jy0o?at_medium=RSS&at_campaign=rss" rel="nofollow noopener" translate="no" target="_blank">http://www.socialpsychology.org/client/redirect.php?from=rss_feed&id=187067&url=https://www.bbc.com/news/articles/cdrkmk00jy0o?at_medium=RSS&at_campaign=rss</a>Source: BBC News - TechnologyNearly 300,000 user conversations with Elon Musk's artificial intelligence chatbot Grok have been exposed in search engine results—seemingly without users' knowledge. Unique links are created when Grok users click to share a transcript of their conversation, but in addition to sharing it with the intended recipient, the button appears to have made the chats searchable online, leading one expert to describe AI chatbots as a "privacy disaster in...URL: <a href="http://www.socialpsychology.org/client/redirect.php?from=rss_feed&id=187067&url=https://www.bbc.com/news/articles/cdrkmk00jy0o?at_medium=RSS&at_campaign=rss" rel="nofollow noopener" translate="no" target="_blank">http://www.socialpsychology.org/client/redirect.php?from=rss_feed&id=187067&url=https://www.bbc.com/news/articles/cdrkmk00jy0o?at_medium=RSS&at_campaign=rss</a>-------------------------------------------------Private, vetted email list for mental health professionals: <a href="https://www.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">https://www.clinicians-exchange.org</a>Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot <a href="https://mastodon.clinicians-exchange.org/@PTUnofficialBot" class="u-url mention" rel="nofollow noopener" target="_blank">@PTUnofficialBot</a>NYU Information for Practice puts out 400-500 good quality health-related research posts per week but its too much for many people, so that bot is limited to just subscribers. You can read it or subscribe at <a href="https://mastodon.clinicians-exchange.org/@PsychResearchBot" class="u-url mention" rel="nofollow noopener" target="_blank">@PsychResearchBot</a>Since 1991 The National Psychologist has focused on keeping practicing psychologists current with news, information and items of interest. Check them out for more free articles, resources, and subscription information: <a href="https://www.nationalpsychologist.com" rel="nofollow noopener" translate="no" target="_blank">https://www.nationalpsychologist.com</a>EMAIL DAILY DIGEST OF RSS FEEDS -- SUBSCRIBE: <a href="http://subscribe-article-digests.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">http://subscribe-article-digests.clinicians-exchange.org</a>READ ONLINE: <a href="http://read-the-rss-mega-archive.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">http://read-the-rss-mega-archive.clinicians-exchange.org</a>It's primitive... but it works... mostly...-------------------------------------------------<a href="https://mastodon.clinicians-exchange.org/tags/psychology" class="mention hashtag" rel="nofollow noopener" target="_blank">#psychology</a> <a href="https://mastodon.clinicians-exchange.org/tags/counseling" class="mention hashtag" rel="nofollow noopener" target="_blank">#counseling</a> <a href="https://mastodon.clinicians-exchange.org/tags/socialwork" class="mention hashtag" rel="nofollow noopener" target="_blank">#socialwork</a> <a href="https://mastodon.clinicians-exchange.org/tags/psychotherapy" class="mention hashtag" rel="nofollow noopener" target="_blank">#psychotherapy</a> <a href="https://a.gup.pe/u/psychotherapist" class="u-url mention" rel="nofollow noopener" target="_blank">@psychotherapist</a> <a href="https://a.gup.pe/u/psychotherapists" class="u-url mention" rel="nofollow noopener" target="_blank">@psychotherapists</a> <a href="https://a.gup.pe/u/psychology" class="u-url mention" rel="nofollow noopener" target="_blank">@psychology</a> <a href="https://a.gup.pe/u/socialpsych" class="u-url mention" rel="nofollow noopener" target="_blank">@socialpsych</a> <a href="https://a.gup.pe/u/socialwork" class="u-url mention" rel="nofollow noopener" target="_blank">@socialwork</a> <a href="https://a.gup.pe/u/psychiatry" class="u-url mention" rel="nofollow noopener" target="_blank">@psychiatry</a> <a href="https://mastodon.clinicians-exchange.org/tags/mentalhealth" class="mention hashtag" rel="nofollow noopener" target="_blank">#mentalhealth</a> <a href="https://mastodon.clinicians-exchange.org/tags/psychiatry" class="mention hashtag" rel="nofollow noopener" target="_blank">#psychiatry</a> <a href="https://mastodon.clinicians-exchange.org/tags/healthcare" class="mention hashtag" rel="nofollow noopener" target="_blank">#healthcare</a> <a href="https://mastodon.clinicians-exchange.org/tags/depression" class="mention hashtag" rel="nofollow noopener" target="_blank">#depression</a> <a href="https://mastodon.clinicians-exchange.org/tags/psychotherapist" class="mention hashtag" rel="nofollow noopener" target="_blank">#psychotherapist</a>

Psychology News RobotDATE: August 22, 2025 at 01:42PM SOURCE: SOCIALPSYCHOLOGY.ORGTITLE: Pharmaceutical Supply Chain Threatened by Weather DisastersURL: <a href="http://www.socialpsychology.org/client/redirect.php?from=rss_feed&id=187068&url=https://www.upi.com/Health_News/2025/08/21/climate-driven-weather-disasters-drug-supply-chain/1991755784002/" rel="nofollow noopener" translate="no" target="_blank">http://www.socialpsychology.org/client/redirect.php?from=rss_feed&id=187068&url=https://www.upi.com/Health_News/2025/08/21/climate-driven-weather-disasters-drug-supply-chain/1991755784002/</a>Source: United Press International - Health NewsClimate change-driven weather disasters like hurricanes, wildfires, and floods pose an immediate threat to the U.S. drug supply chain, a new study finds. For example, hurricanes have triggered shortages of IV fluids. Nearly two-thirds of all U.S. pharmaceutical production plants are located in a county that has had at least one weather disaster during the past six years, researchers reported Wednesday in the Journal of the American Medical...URL: <a href="http://www.socialpsychology.org/client/redirect.php?from=rss_feed&id=187068&url=https://www.upi.com/Health_News/2025/08/21/climate-driven-weather-disasters-drug-supply-chain/1991755784002/" rel="nofollow noopener" translate="no" target="_blank">http://www.socialpsychology.org/client/redirect.php?from=rss_feed&id=187068&url=https://www.upi.com/Health_News/2025/08/21/climate-driven-weather-disasters-drug-supply-chain/1991755784002/</a>-------------------------------------------------Private, vetted email list for mental health professionals: <a href="https://www.clinicians-exchange.org" rel="nofollow noopener" translate="no" target="_blank">https://www.clinicians-exchange.org</a>Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot <a href="https://mastodon.clinicians-exchange.org/@PTUnofficialBot" class="u-url mention" rel="nofollow noopener" target="_blank">@PTUnofficialBot</a>NYU Information for Practice puts out 400-500 good quality health-related research posts per week but its too much for many people, so that bot is limited to just subscribers. 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Psychology News RobotDATE: August 21, 2025 at 04:00PM SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. ** -------------------------------------------------TITLE: Birth control pills reduce the brain’s functional individualityURL: <a href="https://www.psypost.org/birth-control-pills-reduce-the-brains-functional-individuality/" rel="nofollow noopener" translate="no" target="_blank">https://www.psypost.org/birth-control-pills-reduce-the-brains-functional-individuality/</a>A new neuroimaging study suggests that oral contraceptive pills may alter how the brain is functionally organized. Researchers found that oral contraceptives reduce the individuality of brain network patterns and may influence connectivity in circuits associated with emotional regulation. The findings, published in the journal Human Brain Mapping, add to a growing body of research examining how hormonal contraceptives affect the brain.Oral contraceptive pills are used by over 150 million people worldwide and represent one of the most common forms of hormonal contraception. These pills typically contain synthetic versions of the hormones estrogen and progestin, which work together to prevent pregnancy by suppressing ovulation, thickening cervical mucus, and altering the uterine lining. In doing so, they effectively interrupt the natural hormonal cycles that regulate reproductive function. While they are widely regarded as safe and effective, their influence extends beyond the reproductive system. Because sex hormones also interact with brain regions involved in emotion, cognition, and reward, researchers have raised questions about whether oral contraceptives may alter brain function more broadly.Some users report emotional side effects such as irritability, sadness, or mood instability, particularly during the first few months of use. In other cases, oral contraceptives are associated with mood improvements, especially for those with pre-existing hormonal mood sensitivity. Despite these individual differences, researchers still do not fully understand why some people are more vulnerable to adverse emotional effects while others are not.Previous brain imaging studies have suggested that oral contraceptives may alter brain structure and connectivity, including in regions related to emotional regulation, memory, and social processing. However, these studies have often yielded conflicting results. Many relied on observational designs or small, cross-sectional samples, which limits the ability to draw conclusions about causality. To address these gaps, a research team led by Gino Haase of the University of Cambridge and Nicole Petersen of UCLA conducted a randomized, double-blind, placebo-controlled crossover trial to assess whether oral contraceptive pills produce measurable changes in brain network activity and mood within individuals.The researchers recruited 26 women between the ages of 20 and 33 who reported past negative mood symptoms when using hormonal contraceptives. Each participant completed two study arms: one in which they took oral contraceptive pills (30 µg ethinyl estradiol and 0.15 mg levonorgestrel) for 18–21 days, and one in which they took a matched placebo. A full menstrual cycle washout period separated the two arms. During both phases, participants underwent resting-state functional magnetic resonance imaging (fMRI) scans and provided daily self-reports of mood using the Daily Record of Severity of Problems (DRSP).Blood samples confirmed that hormone levels were suppressed during the oral contraceptive phase. The researchers then analyzed brain imaging data to examine functional connectivity—patterns of synchronized activity between different brain regions over time. Unlike structural connectivity, which refers to the physical wiring of the brain through anatomical pathways, functional connectivity reflects how different regions dynamically coordinate with one another, even in the absence of an explicit task.The research team first attempted to replicate specific findings from three prior studies that had identified changes in connectivity between brain regions such as the amygdala, dorsal anterior cingulate cortex, and putamen during oral contraceptive use. These regions are commonly involved in emotion regulation and reward processing.The current study largely failed to replicate those findings. For instance, it did not observe a previously reported increase in connectivity between the amygdala and ventromedial prefrontal cortex. It also did not find expected changes in the dorsal anterior cingulate or parahippocampal connectivity. One partial exception was an increase in connectivity between the putamen and a region of the middle frontal gyrus, though this occurred in the opposite hemisphere compared to earlier findings.The researchers suggest that methodological differences—including sample size, hormonal formulation, or analytic strategies—may account for the lack of replication. These inconsistencies led the team to explore broader, whole-brain effects of oral contraceptive use.To investigate whether oral contraceptives had more diffuse effects on brain networks, the researchers applied a technique known as functional connectome fingerprinting. This method identifies patterns of connectivity that are unique to each individual and can detect changes in those patterns under different conditions.The analysis revealed that oral contraceptive pills caused participants’ brain connectivity profiles to become more similar to one another, reducing between-person variability. While each participant’s brain pattern remained identifiable across conditions, the overall distinctiveness of each person’s functional connectome was lower during oral contraceptive use. This effect was strongest in networks involved in executive control, somatomotor processing, salience detection, and the default mode.This convergence suggests that oral contraceptive pills may exert a normalizing or dampening effect across the brain’s functional architecture. Rather than targeting specific regions, the influence appears to be more global—altering how entire networks are configured and reducing the brain’s functional individuality.In addition to these network-wide shifts, the researchers also found evidence linking brain changes to self-reported mood symptoms. Participants experienced more negative affect during the oral contraceptive phase, as measured by the DRSP. The researchers identified 13 specific connections between brain regions that significantly correlated with increases in negative mood.These connections involved areas such as the frontal pole, superior frontal gyrus, posterior cingulate cortex, and precuneus—all regions known to play roles in emotional processing, self-referential thought, and regulation of internal states. The findings suggest that oral contraceptive-related changes in these circuits may underlie the mood symptoms reported by some users.Rather than pointing to a single brain region responsible for these effects, the results support the idea of a distributed, hormone-sensitive network that may be involved in emotional changes during hormonal transitions—such as menstruation, postpartum periods, or contraceptive use.But the study, like all research, has limitations. The sample size was relatively small (26 participants), which may limit generalizability and increase the likelihood of false positives or missed effects. Only one formulation of oral contraceptive pills was tested, so the results may not apply to other hormonal combinations. The study measured brain activity at rest, meaning it did not capture how oral contraceptives affect the brain during emotional or cognitive tasks.Additionally, the intervention lasted only a few weeks. It remains unknown whether longer-term use would amplify, reduce, or change the nature of these connectivity shifts. Finally, although associations between brain connectivity and mood were observed, the study cannot establish a definitive causal pathway linking them.Despite these limitations, the study suggests that oral contraceptive pills can produce measurable and widespread changes in brain connectivity. These changes are not limited to isolated regions but span across multiple major brain networks. The results also suggest that oral contraceptive pills may reduce the uniqueness of individual brain network patterns, a finding with implications for both research and clinical care.The researchers propose that future studies could build on these findings by enrolling larger and more diverse samples, testing additional formulations, and using multivariate tools like connectome fingerprinting to better understand how hormonal contraceptives interact with brain function and behavior. They also suggest that the network of brain regions identified in this study could serve as a candidate for investigating mood changes across different hormone-sensitive conditions.The study, “Effects of Oral Contraceptive Pills on Brain Networks: A Conceptual Replication and Extension,” was authored by Gino Haase, Jason Liu, Timothy Jordan, Andrea Rapkin, Edythe D. 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